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Recent investigations have revealed that ferroptosis involves alterations in iron metabolism regulatory proteins (e.g., GPX4, TfR1), accumulation of lipid peroxidation products, and dysregulation of antioxidant systems, particularly the nuclear factor E2-related factor 2 (Nrf2)/glutathione peroxidase 4 (GPX4) pathway (8, 9)

Accumulation with age of vesicular GTPases involved in endocytosis (Rab7) and lysosomal degradation (Arl8b) Small GTPases drive essential processes to clear aggregated or misfolded proteins in the cell by the process of autophagy [7]

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This delayed presentation may relate to cumulative receptor activation and gradual changes in neural sensitivity thresholds

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Injection site issues Local reactions at injection sites occasionally cause concern